The investigation into psychological stress is crucial for understanding its impact on human functioning. This report details an experiment designed to measure the physiological and subjective responses to a standardized stressor. The primary hypothesis posited that participants exposed to a challenging cognitive task would exhibit significantly higher levels of cortisol, heart rate, and self-reported anxiety compared to a control group.
Participants were recruited from a university undergraduate population, with 40 individuals (20 male, 20 female) volunteering. They were randomly assigned to either the experimental (stress induction) group or the control group. The stress induction task involved a timed arithmetic test requiring rapid problem-solving, coupled with negative social feedback delivered by the experimenter. The control group engaged in a similar duration of a passive reading activity. Salivary cortisol samples were collected at baseline, immediately post-task, and 30 minutes post-task. Heart rate was continuously monitored throughout the procedure. Participants also completed the State-Trait Anxiety Inventory (STAI) immediately after the task.
Analysis of the data supported the hypothesis. The experimental group showed a marked increase in salivary cortisol levels from baseline to post-task (M=15.2 ng/mL, SD=4.1) and this elevation remained significant at the 30-minute mark (M=12.8 ng/mL, SD=3.5), whereas the control group exhibited minimal fluctuation (Baseline M=8.5 ng/mL, SD=2.3; Post-task M=9.1 ng/mL, SD=2.5; 30-min M=8.8 ng/mL, SD=2.4). An independent samples t-test revealed a significant difference in cortisol levels between the groups at both post-task and 30-minute intervals (t(38) = 5.89, p < .001; t(38) = 4.21, p < .001).
Similarly, heart rate data indicated a significant difference. The experimental group's average heart rate increased from 72 bpm at baseline to 95 bpm immediately post-task, whereas the control group's heart rate remained relatively stable, averaging 70 bpm at baseline and 74 bpm post-task. A repeated measures ANOVA on heart rate data showed a significant interaction effect between group and time (F(2, 76) = 8.92, p < .001), confirming that the stress induction led to a greater increase in heart rate.
Subjective reports also aligned with physiological findings. The STAI-State scores for the experimental group were substantially higher (M=48.5, SD=8.2) than for the control group (M=30.1, SD=6.5). An independent samples t-test confirmed this difference was statistically significant (t(38) = 7.15, p < .001). These findings collectively demonstrate the effectiveness of the cognitive and social feedback manipulation in inducing a measurable stress response.
The results of this experiment corroborate existing literature on stress response. The physiological indicators, namely elevated cortisol and heart rate, alongside increased self-reported anxiety, provide a comprehensive picture of the acute stress reaction. The controlled environment and standardized procedures allowed for a clear attribution of these changes to the stress induction task. Future research could explore individual differences in stress susceptibility and the long-term effects of such acute stressors.